LaTeX templates and examples — Reports

Template para relatórios de alunos da Universidade Federal do Pará

This is a template for documents of the FGUMA online courses (www.fguma.es).

A bare-bones template for writing Linguistics papers at Pomona College. It may also be useful to linguists/linguistics students at other places. Includes links to our quick reference guide as well, which has more detailed instructions on formatting for linguistics papers.

This template is a fork from an earlier version of the Purdue University Senior Design Report Template. It is modified and edited by Dr. Abdallah S. Abdallah, an assistant professor of electrical and computer engineering at Penn State Erie - The Behrend College

This is a template style useful to make assignments, progress reports, and thesis submissions for Indian Institute of Engineering Science and Technology, Shibpur.

Template originalmente concebido para a confecção de relatório de atividades no DEE. Pode ser utilizado para outras finalidades, além de ser permitido modificá-lo irrestritamente.

This is a self-generated LaTeX template for students in The University of Sydney (USYD) to deal with coursework assignments.

Style based on MT Summit 2019 template

Bacterial cells can have DNA damage due to transcriptional error, or through the effect of an antibiotic. The SOS response is a bacterial cell program for coping with DNA damage, in which the cell cycle is arrested, and DNA repair is induced. The repairs have high probability in leading to mutagenesis in the bacteria, which can lead to antibiotic resistance. The RecA protein in bacteria is responsible for the activation of the SOS response; therefore, making it a target for inhibition. I elected to use the ubiquitination system, natively used for apoptosis, as a means of targeted degradation of the RecA protein in bacteria prone to mutations. Polyubiquitination of misfolded proteins leads to the breaking down of the protein with the aid of proteasomes, which break down unnecessary proteins through a chemical reaction known as proteolysis. Using random forest-predictors, I determined a statistically high likelihood of ubiquitination of the RecA protein in MRSA, Tuberculosis, and other high risk bacterial infections. I hypothesized that I could foster ubiquitin-tagging on RecA by forcing the protein to misfold. Chaperones are proteins which interact with each other to prevent specific sets of proteins from misfolding. CHIP (C terminus of HSC70-Interacting Protein) is a biomolecule that inhibits interactions between the chaperones of RecA. Adding CHIP, ubiquitin, and 26s proteasomes into the bacterial system, should theoretically lead to the degradation of the RecA protein inside the bacteria. I tested my hypothesis by conducting an assay for monitoring CHIP-mediated ubiquitination, and conducted analysis on the assay using SDS- Page gel electrophoresis, and Western-blotting. The resulting data showed signs of polyubiquitination on the RecA protein, with chains of five or more ubiquitin, showing high drug potential. Adding an antibody drug conjugate, containing all the necessary components of a CHIP-mediated ubiquitination reaction, to common antibiotics can lead to the inhibition of bacterial mutagenesis, and higher antibiotic drug potency.